Tebuconazole is a fungicide for the control of smuts, bunt, seed rots and seedling blights on barley, oats and wheat as a seed treatment and for the control of Fusarium Head Blight on wheat as a post-emergent treatment.
1.Number of Tebuconazole Applications for Maximizing Disease Control
and Yield of Peanut in GrowersĄŻ Fields in Alabama.
Disease and yields of peanut were monitored in growersĄŻ fields treated with zero, one, two, three, and four applications of tebuconazole. Yields ranged from 1,918 to 6,891 kg/ha and averaged 4,319 kg/ha over eight locations in 3 study years. Defoliation caused by peanut leaf spots (Cercospora arachidicola and Cercosporidium acutatum) and incidence of southern stem rot (Sclerotium rolfsii) were inversely related to number of tebuconazole applications, while yield was directly related to number of tebuconazole applications. Average yields with zero, one, two, and four tebuconazole applications (each application at 0.25 kg a.i./ha) were 3,609, 4,088, 4,526, and 4,868 kg/ha, respectively. Data from each location were best modeled by quadratic equations allowing determination of numbers of tebuconazole applications for minimal disease and maximal yields. Calculated spray numbers indicate that between three and four applications of tebuconazole is best for disease control and yield maximization. For detail information for this article, please visit here.
Effects of tebuconazole on morphology, structure, cell wall components and trichothecene production of Fusarium culmorum in vitro.
The effects of tebuconazole, a systemic fungicide, on the morphology, structure, cell wall components and toxin production of Fusarium culmorum were investigated in vitro . Treatment was by application of four filter paper strips (0.75 cm ?nbsp;5.0 cm) soaked in 20 ”g ml -1 fungicide placed around a point inoculum in Petri dishes. Mycelial growth was strongly inhibited by fungicide treatment. Scanning electron microscopic observations showed that the fungicide caused irregular swelling and excessive branching of hyphae. The morphological changes induced by the fungicide at the ultrastructural level included considerable thickening of the hyphal cell walls, excessive septation, the formation of the incomplete septa, extensive vacuolisation, accumulation of lipid bodies and progressing necrosis or degeneration of the hyphal cytoplasm. Non-membrane inclusion bodies were often detected in the hyphal cytoplasm. Furthermore, the formation of new hyphae (daughter hyphae) inside collapsed hyphal cells was common following treatment. The daughter hyphae also displayed severe alterations such as irregular thickening of the cell walls and necrosis of the cytoplasm. Using cytochemical techniques, the labelling densities of chitin and -1,3-glucan in the cell walls of the fungicide-treated hyphae were more pronounced than in those of the control hyphae. Moreover, immunogold labelling with antiserum against deoxynivalenol (DON) revealed that Fusarium toxin DON was localized in the cell walls, cytoplasm, mitochondria and vacuoles of the hyphae from the control and the fungicide treatment, but the labelling density in the fungicide-treated hyphae decreased dramatically compared with the control hyphae, indicating that tebuconazole reduced Fusarium toxin production of the fungus.
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Oral acute oral LD50 for rat 3776mg/kg; Acute dermal LD50 for rat 2011 mg/kg; Acute inhalation LC50 for rat 2.51 mg/l.
Technical product is Colourless to light brown powder. Solubility in water Dispersible; soluble in acetone. Melting Point: 102.4-104.7 Ąă C